Neurodegeneration and Neuroprotection in Parkinson's Disease - download pdf or read online

By C. W. Olanow, Peter Jenner, Moussa Youdim

ISBN-10: 0080537413

ISBN-13: 9780080537412

ISBN-10: 0125254458

ISBN-13: 9780125254458

Neuroscience views offers multidisciplinary studies of themes in a single of the main divers and speedily advancing fields within the existence sciences. even if you're a new recruit to neuroscience, or a longtime specialist, glance to this sequence for 'one-stop' assets of the old, physiological, pharmacological, biochemical, molecular organic, and healing points of selected study components.

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1988a)J. Neurosurg. 68, 456-461. , Yonkers, RA. M. (1988b) Eur. J. Pharmacol. 147, 299-303. L. & Jacobsen, EJ. (1990) Stroke 21 (Suppl. III), 83-87. Hallgren, B. & Sowander, R (1958)J. Neurochem. 3, 41-51. Halliwell, B. (1978) FEBS Lett. 92, 321-326. Halliwell, B. (1987) FASEBJ. 1,358-364. Halliwell, B. (1989) Free Radical Res. Commun. 5, 315-318. Halliwell, B. (1992)J. Neurochem. 59, 1609-1623. Halliwell, B. C. (1984) Lancet i, 1396-1398. Halliwell, B. C. (1985) TrendsNeurosci. 8, 22-26. Halliwell, B.

4 Batten's disease The neuronal ceroid-lipofuscinoses (NCLs) are a group of recessively inherited neurodegenerative lysosomal storage diseases which have a particularly high incidence in the Nordic countries. In all types of NCLs there is a widespread deposition of ceroid and lipofuscin pigments in the body, which has, in the past, led to oversimplistic claims that lipid peroxidation was a key event in disease progression. , 1990). , 1988). , 1988). 5 Abetalipoproteinaemia In abetalipoproteinaemia, patients have a rare inborn error of lipid metabolism in which dietary fat is ingested and absorbed, but not transported out of the intestinal mucosal cells, because of an inherited inabili~ to synthesize apoprotein B.

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Neurodegeneration and Neuroprotection in Parkinson's Disease (Neuroscience Perspectives) by C. W. Olanow, Peter Jenner, Moussa Youdim

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