By Gerald Fenichel MD
Thoroughly up to date, completely referenced, and good illustrated, the fourth version of Gerald Fenichel's vintage assessment, Neonatal Neurology walks you thru the newest advances within the medical analysis and administration of neurological issues within the child.
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During this elegantly written and accomplished background, we meet people who suffered with a number of sclerosis within the centuries sooner than the illness had a reputation, together with blessed Lidwina of Holland, who took pleasure from her distress, believing that she used to be despatched to simply accept affliction for the sins of others; Augustus, grandson of George III and cousin of Queen Victoria, whose case exhibits how a person with entry to the easiest of treatment of the age used to be understood and controlled; and Heinrich Heine, the good German poet, who additionally had entry to all clinical companies that have been on hand, yet who advanced into his bed grave in twenty years, conscious of the lack of actual skill whereas nonetheless in a position to compose nice poetry to the top.
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Extra resources for Neonatal Neurology (Fourth Edition)
Perinatal hypoxic-ischemic thalamic injury: Clinical features and neuroimaging. Ann Neurol 1998;44:161–166. Scheuerle AE. Incontinentia pigmenti. (27 March 2003) In: GeneClinics: Medical Genetics Knowledge Base. [database online] University of Washington, Seattle. org Volpe JJ. Brain injury in the premature infant-neuropathology, clinical aspects, pathogenesis and prevention. Clin Perinatol 1997;24:567–587. Volpe JJ. Neurology of the Newborn, 4th edn. Philadelphia: WB Saunders, 2001;185–186. Wyatt JS.
Reduced fetal movement may occur when neuronal degeneration begins in utero. Affected newborns have generalized weakness involving proximal more than distal muscles, hypotonia, and areflexia. Newborns that are hypotonic in utero and weak at birth may have difficulty adapting to extrauterine life and experience postnatal asphyxia and encephalopathy. Most breathe adequately at first and appear alert despite the generalized weakness because facial expression is relatively well preserved and extraocular movement is normal.
Characteristic of the third phase is apparent improvement with normalization of tone. This may cause second thoughts about the accuracy of the diagnosis, but the improvement is short-lived. Evidence of neurological dysfunction begins to appear toward the end of the second month, and the symptoms become progressively worse throughout infancy. In premature newborns, the clinical features are subtle and may lack the phases of increased tone and opisthotonos. The typical clinical syndrome after the first year includes extrapyramidal dysfunction, usually athetosis, which occurs in virtually every case (see Ch.
Neonatal Neurology (Fourth Edition) by Gerald Fenichel MD